Do you have high blood pressure?
If so, don’t worry (too much)!
While blood pressure, or hypertension, has recently been found to be the most important risk factor for premature death , there is good news.
We now know that a poor diet and a sedentary lifestyle is the primary cause of hypertension. And given the fact that many of us are afflicted with this unhealthy condition, it's good to know that we can actually do something about it!
It’s estimated that the majority of Americans over the age of 60 have clinical hypertension. What's interesting is that if you were to compare that to modern hunter gatherers of the same age, this number is drastically lower .
What's the difference? Modern hunter gatherers still incorporate the types of food into their diets that our bodies were evolved to process in a healthy manner AND they live an active lifestyle – full of exercise.
In this article, we’re going to discuss how to tackle high blood pressure naturally. It’s certainly worth trying at least; before you pump your body full of prescription drugs that only treat the symptom of hypertension, and not the underlying cause. As we’ll discuss later, these drugs also come with their own, oftentimes dangerous, set of symptoms.
What Is High Blood Pressure and How Is It Measured?
High blood pressure is an unfortunately common condition where the force of your blood against your artery walls is high enough that it can eventually cause health problems like heart disease.
Blood pressure is physically caused by two factors:
- The amount of blood your heart pumps and
- The amount of resistance to blood flow in your arteries.
So, the more blood your heart pumps and the narrower your arteries, the higher your blood pressure.
Luckily, blood pressure is easy to measure. When you read your blood pressure, it’s expressed as a measurement with two numbers, in units of millimeters of mercury (mm Hg), expressed as a fraction. 120/80 or “120 over 80”, for example.
The top number is your “systolic pressure” and is a measurement of the amount of pressure in your arteries when your heart is contracting.
The bottom number is your “diastolic pressure” and is a measurement of your blood pressure between heart beats.
According to the American Heart Association, high blood pressure is diagnosed when your systolic pressure is above 120 mm Hg or your diastolic pressure is above 80 mm Hg .
Why Do We Care?
Symptoms of high blood pressure can include nosebleeds, headaches and shortness of breath. The majority of people are asymptomatic and, unfortunately, you can live with high blood pressure for years and not have any symptoms – until you actually get your blood pressure measured by a clinician. Even if you don’t have symptoms, damage to your blood vessels and your heart continues. If you don’t control your blood pressure, your risk of serious and deadly health problems including heart attack and stroke [4, 5].
Rather than address the underlying causes of high blood pressure, conventional physicians prescribe a number of various drugs to treat hypertension. These include:
Diuretics – also called water pills – trigger your kidneys to remove sodium and water from your body by increasing your rate of urination. This eases the pressure on your arteries by lowering your blood pressure. Diuretics can include:
- Diuretics are the first and most commonly prescribed medication to treat high blood pressure. Not only do they decrease fluids, but they also cause your arteries to relax. Thiazides are often prescribed with other drugs to lower blood pressure.
- Loop diuretics. Loop diuretics work by interfering with the movement of salt and water across certain cells in your kidneys – forcing them to pass more fluid. As your kidneys pass more fluid, less remains in your blood stream thereby reducing your blood pressure.
- Potassium sparing diuretics. Potassium sparing diuretics are commonly used in combination with other types of diuretics to ensure that your potassium levels remain at a safe level.
While diuretics are generally safe, they do have some side effects, including :
- Hyperkalemia and hypokalemia (too much or too little potassium).
- Hyponatremia (low sodium).
- Muscle cramps
Beta blockers are used widely for high blood pressure, either alone or in combination with other medicines. Beta blockers act by lowering your heart rate and are typically prescribed to people who also have angina, heart failure or who’ve already had a heart attack. There are several side effects associated with beta blockers, including :
- Trouble breathing.
- Swelling of the face, lips, tongue and throat.
- Very slow heart rate.
- Swelling in the legs or feet.
- Trouble sleeping.
Angiotensin-converting-enzyme (ACE) inhibitors are pharmaceutical drugs used primarily for the treatment of hypertension. ACE inhibitors work by preventing your body from creating a hormone known as angiotensin II. Angiotensin II has three main effects, including:
- Constriction of blood vessels.
- Re-absorption of water by your kidneys.
- Release of aldosterone, another hormone that causes water re-absorption by the kidneys.
To summarize, ACE inhibitors essentially relax blood vessels and helps to reduce the amount of water re-absorbed by the kidneys . Both of these mechanisms work to lower your blood pressure. Side effects of ACE inhibitors include:
- Hypotension (low blood pressure)
- Dry cough
- Swelling of the lips, eyes and tongue.
- A decline in kidney function.
Calcium Channel Blockers
Calcium channel blockers (also called calcium antagonists) are another commonly prescribed medicine to reduce blood pressure. They work by relaxing and widening the blood vessels by affecting muscle cells in the arterial walls. There are both short-acting – which work quickly but only last a few hours – and long-acting – which are released slowly but provide a longer effect . Side effects of calcium channel blockers include:
- Foot and lower leg swelling
- Heart palpitations
So now that we know a little more about the drugs that conventional practitioners prescribe to treat the symptom of high blood pressure, let’s discuss the lifestyle factors that actually contribute to the underlying cause. As I mentioned before, high blood pressure is virtually nonexistent in modern hunter gatherers. While diet, sleep, stress and whether you smoke all contribute to cause high blood pressure, let’s focus in on diet because that packs the biggest bang for the buck (smoking arguably rivals diet when it comes to high blood pressure, but let’s assume, for this discussion, that you’re a non-smoker).
So, what is it in our diet that causes high blood pressure?
Let’s dig a little deeper into the diet-blood pressure connection and focus on sugar and mineral and nutrient imbalances.
Our modern diet is full of sugar – from fructose in fruit juices to high fructose corn syrup in soda and sweetened cereal to sucrose in candy – it’s everywhere in our modern, processed foods. In fact, a lot of this sugar is “hidden” in some foods we don’t even realize or think about – baked goods made from white, “enriched” flour, alcoholic beverages like beer and wine, and in condiments and even crackers. The list goes on and on. A recent study by Louisiana State University looked at over 800 people with some form of hypertension in an effort to find out how sugary drinks affected blood pressure. The study found that people who drank one less serving of a sugary drink per day had a measurable decline in blood pressure after a year and a half .
Mineral and Nutrient Imbalances
A healthy diet plays a major role in blood pressure control. Specifically, obtaining the right amounts of sodium, potassium, magnesium and calcium in our diets are crucial to maintaining low blood pressure. Too often, however, our modern diets lack in in some minerals – especially potassium, magnesium and calcium – and too high in others – namely sodium. Throwing these mineral balances out of whack with a poor diet full of processed foods will do a number on your blood pressure.
The Natural Fix For High Blood Pressure
So, how do we attempt to fix high blood pressure without resorting to prescription drugs?
I would maintain it’s quite simple, really. Maybe as easy as 1-2-3.
- If you smoke, quit. This is really a no brainer as smoking and the use of tobacco products are extremely poor for your long-term health. You all should know this so I won’t waste too much time extolling the virtues of a smoke-free lifestyle.
- Clean up your diet. How do we avoid the sugar and mineral and nutrient imbalances that play a huge role in high pressure? I would recommend adopting a paleo, or ketogenic or other ancestral diet. Eat meat and vegetables. Limit your dairy to start and avoid all processed foods, alcohol and sugar of any kind. There are a ton of great resources online on how to get started but the main advice is: stick to the outside of the grocery store. Give it a shot for 30 days. Not only will your blood pressure go down, you’ll lose weight and, if you make it through the first week, you’ll feel so good you’ll wonder why you haven’t been eating this way your entire life.
- Work on your sleep and exercise. Finally, if you’ve made it through steps one and two, take a look at your sleep habits and start to adopt some easy to stick to exercise routine. Try starting with a 30 minute walk each morning and going to bed at the same time every night (even on the weekends).
Drastic lifestyle changes can be tough to implement, but I would maintain that they are entirely worth it. Especially compared to the high blood pressure “band-aids” that are prescription drugs. The potential side effects from these blood pressure medications we talked about before are just no fun. I challenge you to give yourself a couple months to try making it through the steps I outlined above. If you can, I can almost promise you’ll not only feel great, but you’re blood pressure will drop measurably.
If you have any questions on any of the information I presented in this article, or to talk to me about a consultation to try to implement some of the natural high blood pressure fixes I talked about, contact me today.
Statin Drugs: Are They Worth the Risks?
Statins (cholesterol lowering medications) are one of the most commonly prescribed medications in America. Approximately one in every four adults over age 40 take these drugs to lower their cholesterol levels.
It is no secret that statin drugs are recommended left, right and center to people with elevated cholesterol levels, to those with existing heart problems and even to healthy people as a primary form of prevention.
We have been led to believe that statins lower cholesterol levels and by extension reduce the risk of heart disease and stroke. While statins indeed reduce cholesterol levels, this is only a part of the entire puzzle. The problem is: the proof that this ‘cholesterol lowering outcome’ prevents any major cardiovascular event such as heart attack or stroke, simply doen’t exist. Cholesterol levels, unless they are drastically off the charts, are not a very strong predictor of your heart disease risk.
Another fact is that the benefits of statins are mostly overstated, whereas the associated risks are often underreported.
How do statin drugs works?
Your liver produces cholesterol through a pathway called the mevalonate pathway – that involves an enzyme HMG-CoA reductase. Statin drugs block the production of this enzyme and lower the cholesterol production in the liver. The picture below shows this process.
However, things don’t end here. Besides cholesterol, the mevalonate pathway (with the enzyme HMG-CoA reductase present at the base of it all) is also involved in the production of other non-sterol compounds such as CoQ10 (ubiquinone), dolichols, heme-A, selenium containing proteins, sex hormones, corticosteroids, bile acids and vitamin D. 
So, when you are using statin drugs, you are not only lowering your cholesterol levels (the simmering debate is whether we really need to?) but also disrupting the production of so many other compounds that are essential for cell growth, and maintain heart, muscles, brain and immune health.
Do statins drugs really work?
Well, statin drugs do what they are meant to do; they lower your cholesterol levels. But does this mean you are getting any healthier? Or that your risk of getting a heart disease or stroke is decreased? That will be one wrong assumption; with no real scientific backing at all.
In my previous article, we discussed the importance of cholesterol in the body and that lowering cholesterol has no real significance on your heart health.
On the other hand, there is a plenty of evidence that statin drugs come with many serious side effects. We will talk about these side effects in details, but first, it is time to address the most controversial question.
Do statins really prevent heart disease?
First, it is important to know that your cholesterol levels play a minor role, if at all, when it comes to your risk of developing heart disease or getting a heart attack. Chronic inflammation (prompted by oxidative damage, stress and excessive consumption of sugar and processed foods) contribute much more to this risk.
We blame cholesterol because it is found at the site of inflammation, injury and damage but that is only because it is there to help in its anti-oxidant and anti-inflammatory capacity. Cholesterol is also easy to blame because you can significantly lower it by using medications.
Secondly, evidence suggests that statins may actually increase your risk of developing atherosclerosis and heart problems.
Research and clinical trials prove that taking statins is nothing sort of a gamble – only favorable in some patients and under narrow circumstances. That brings us to the use of statins in primary prevention and secondary prevention.
Statin drugs in primary prevention: In people without any pre-existing heart condition
A study examined the effect of statins in people who have never suffered a heart attack or stroke; which is mostly the case as statin drugs are commonly prescribed at the first sign of elevated cholesterol.
Not surprisingly, the analysis found that statins do lower cholesterol in most of these people. But only a very few people will dodge a cardiovascular event (a heart attack or a stroke) because of the cholesterol lowering outcome. More specifically, 
- 1 in 60 (1.6%) prevented a heart attack
- 1 in 268 (0.4%) prevented a stroke
- 1 in 67 (1.5%) developed diabetes
- 1 in 10 (10 %) suffered from muscle damage
In addition, the drugs don’t show any benefits whatsoever when it comes to extending the lifespan. A 2010 meta-analysis of the clinical trials examined whether stains have any effect on longevity in a high-risk primary prevention scenario. The study found that statin therapy does not significantly reduce all-cause mortality risk among high-risk people without any with clinical history of coronary heart disease. 
Statin benefits are obviously overstated!
In 2015, a study published in Expert Review of Clinical Pharmacology indicated that both the safety and life-saving ability of statin drugs have been exaggerated. The researchers stated that statins effectively reduce cholesterol levels, but do not offer any impressive cardiovascular disease outcomes, also adding that statin researchers use ‘statistical deception' to embellish claims about their effectiveness. 
For example, statins actually benefit only 1% of the population; meaning these drugs will prevent a heart attack in only one out of 100 patients. This is the absolute risk. But this figure is certainly not presented as is, but by using another statistic called the ‘relative risk’. This results in creating an illusion that statins prove effective in 30-50% of the population.
The study authors, Dr. David M. Diamond and Dr. Uffe Ravnskov explained further “In the Jupiter trial, the public and healthcare workers were informed of a 54 percent reduction in heart attacks, when the actual effect in reduction of coronary events was less than 1 percentage point.”
“In the ASCOT-LLA study, which was terminated early because it was considered to have such outstanding results, there were heart attacks and deaths in 3% of the placebo (no treatment) group as compared to 1.9% in the Lipitor group. The improvement in outcome with Lipitor treatment was only 1.1 percentage point, but when this study was presented to the public, the advertisements used the inflated (relative risk) statistic, which transformed the 1.1% effect into a 36% reduction in heart attack risk.” 
John Abramson, MD (of Harvard Medical School and author of Overdosed America), and James Wright, MD (of University of British Columbia) analysed 8 clinical trials on the effect of statins versus placebo. They reported in the journal Lancet that the absolute risk reduction of 1.5% is insignificant.  The analysis further suggested that:
- Statins should not be prescribed in women of any age or for men older than 69 years (as a primary prevention.)
- Men considered high risk and aged 30-69 years should be counselled that about 50 patients need to be treated for 5 years to prevent one cardiovascular event.
Clinical data do not support the use of statin therapy in primary cardiovascular prevention. On the other hand, plenty of evidence points to the fact that statins actually “augment cardiovascular risk in women, patients with Diabetes Mellitus and in the young”. 
So, the question is whom do the statins work for anyway?
Statin drugs in secondary prevention: In people with pre-existing heart condition
Studies suggest that statins are effective in people who already have had heart attacks or cardiovascular events. However, even for this set of population the efficacy of statins is not very remarkable.
As this analysis showed, 5 years of daily statin treatment could only save 1 out of 83 people. The therapy prevented stroke in 1 out of 125 people and prevented non-fatal heart attack in 1 out of 39 subjects. At the same time, use of statins over this period caused diabetes in 1 out of 5 people and muscle damage in 1 out of 10 people. 
But how do you know where to draw the line? Let’s simplify this.
Statins can be beneficial in some patients with pre-exiting history of developing coronary heart disease, that is when used a secondary prevention. This is where the potential risk is quite high. But if you fall in the low-risk category with no prior history of a heart problem, proceed with caution.
Again, it pays to remember that cholesterol is not a causative factor in heart disease and trying to lower it by using statins, especially in elderly and women, is simply unwarranted. Consult an expert healthcare professional to help you understand your benefits and associated risks, and decide if the gamble is worth.
Statins are beneficial, even needed, if you have familial hypercholesterolemia
Familial hypercholesterolemia is a serious genetic condition where the affected person has high levels of blood LDL cholesterol. Here, the body is not naturally equipped to remove excess LDL from the blood. The condition is present at the time of birth and can cause heart attack and other cardiovascular problems at quite an early age.
Side effects of statins: Worth the risk?
Statins exert a wide range of adverse effects that more than offset the modest benefits.
Revisiting the 2015 study by Diamond and Ravnskov, the authors stressed that cholesterol lowering consequence of statins is often associated with an increased risk of cancer. They also highlighted that most statin trials are concluded within a span of two to five years. This period is not long enough to record the development of most cancers.
Coming back to the question: Do Statins Really Prevent Heart Disease? On the contrary. Let’s find out.
- Statins and heart disease risk
A 2015 study published in Expert Review of Clinical Pharmacology showed that statins may actually trigger atherosclerosis and heart failure. The study highlighted various mechanisms to show statins may aggravate your heart problems and concluded that statins work as mitochondrial toxin that “impair muscle function in the heart and blood vessels through the depletion of coenzyme Q10 and ‘heme A', and thereby ATP generation.”  Let’s take a look.
- Statins supresses CoQ10 production
Statin drugs target mevalonate pathway to control cholesterol production. But this important pathway is also involved in the synthesis of CoQ10 – a coenzyme present in every cell of the body.
CoQ10 plays an indispensable role in the production of ATP, the energy currency, in the mitochondria. Without CoQ10, our body won’t be able to produce any bit of energy. The co-enzyme is also a powerful anti-oxidant and protects the arteries from oxidative damage and ensuing inflammation. It is easy to see how these properties translate into heart health benefits?
Your heart needs the maximum energy funding to continue beating nonstop – pumping oxygen and nutrient-rich blood across the body. This is the precise reason why heart has the highest concentrations of CoQ10 as compared to any other organ in your body. Now, statin drugs deplete your body of CoQ10. Low CoQ10 levels, especially in the heart, contribute to the onset and progression of congestive heart failure (CHF) and heart attack. More on the role of CoQ10 in our next article.
Statins have adverse effects on skeletal muscle – ranging from mild muscle pain, muscle fatigue to more serious rhabdomyolysis. Statin drugs inhibit the production of CoQ10 levels, an action that impairs the energy production in the muscles. This causes muscle weakness and fatigue.
A 2013 study concluded that musculoskeletal conditions, arthropathies, injuries, and pain are more common in people who are on statins than non-users. The study explained that among other factors, the blocking effect of statins on the synthesis of COQ10 as well as the selenoproteins may be at play here.  Selenoproteins such as glutathione peroxidase play a pivotal role in averting oxidative damage in the muscle tissue.
One rare, but extremely dangerous, side effect of statins is a condition called rhabdomyolysis. In this condition, damaged muscle tissues break-down and release a protein called myoglobin into the bloodstream. Too much of myoglobin in the blood can cause damage to the kidneys. The most telling symptoms of rhabdomyolysis include intense muscle pain throughout the body, muscle weakness, dark colored urine and general feeling of being sick. The risk of rhabdomyolysis increases with the increased dose of statins.
- Statins block vitamin K2 synthesis
Statins inhibit the production of vitamin K2. The vitamin is known to mobilize calcium into the bones and keep it away from arteries and soft tissues – thus preventing calcium deposition and build-up of plaque in the arteries.
Vitamin K2 activates matrix Gla-protein, that prevents calcification in the arteries.  Vitamin K2 also activates osteocalcin – a protein that holds onto calcium and directs it towards the bones – making bones stronger and less prone to fracture.
- Statins and cognitive functions decline
It is simple. Your liver makes cholesterol because the body needs it for so many things (to make vitamin D, bile acids and all kinds of important hormones).
When your cholesterol levels drop too low, as with long-term and high dose statin users, your body would seek it from other cholesterol-rich sources, for example the brain. Your brain contains about 25 % of the cholesterol in the body. It plays a critical role in the transmission of neurotransmitters and facilitates communication between the neurons, which helps us to think and affects how well we learn new things and make memories. When cholesterol dips too low in the brain, a lot of patients on statins suffer memory loss and decline in their cognitive faculties.
We know that statins work by reducing cholesterol production in the liver. A 2009 research by Yeon-Kyun Shin, an Iowa State University scientist, demonstrated that statins may also keep the brain from synthesising cholesterol.
Shin tested how the release of neurotransmitters by the brain cells is affected both in the absence and presence of cholesterol. When he measured the protein function in the brain with cholesterol in attendance, there was a fivefold increase in the protein function. Shin further explained that cholesterol alters the shape of the protein to stimulate thinking and memory. 
In 2009, a research published in the Journal of Lipid Research concluded that “the long-term effects of statin therapy could lead to transient or permanent cognitive impairment in patients who already have low levels of brain cholesterol.” 
This is a huge cause of concern in the elderly because with age, there is a lot less production of cholesterol in the brain already. In addition, cholesterol as well as phospholipid levels are reduced in the patients with Alzheimer’s Disease. The study showed that statins influence the expression of genes involved in neurodegenerative processes and suggested a potential mechanism that triggers statin-induced cognitive impairment in vulnerable patients.
- Statins and risk of diabetes
Statins increase your risk of diabetes through many mechanisms.
Studies show that long-term statin therapy increases the risk of new-onset diabetes, possibly by damaging the functions of pancreatic beta cell and by reducing insulin sensitivity.   In addition, statins elevate blood sugar levels. The drugs also inhibit the production of CoQ10, a nutrient that helps to control blood sugar levels.
- Statin and cataracts
Cataract, the cloudiness in the eye lens, is a leading cause of reduced or blurred vision among the elderly. It is primarily caused by oxidative damage induced by free radicals. Studies show that statins increase the risk of developing cataracts. 
A recent 2016 study hinted at the possibility that increasing use of statins could be contributing to “rising rates of cataract surgery.” 
So, are statins any good?
- Yes, statins do lower cholesterol levels but this outcome does not translate into any significant cardiovascular protection, especially in people without any history of coronary heart disease.
- Statins are effective in reducing heart attacks and death (from heart disease) in people with existing heart problems; especially in middle aged males.
- Statins shouldn’t be prescribed to elderly people or to women.
- People with active liver disease should not be prescribed statins.
- Pregnant and breast-feeding women should not take statins.
- Statins are extremely dangerous for children in particular, except in familial hypercholesterolemia (a rare genetic condition).
- Statins may have anti-inflammation properties but they don’t influence the size of cholesterol particles.
So, what should you do when you are diagnosed with elevated cholesterol? Making lifestyle changes is a safer and much more effective approach over pharmaceutical interventions. This is the single most important method to reduce the oxidative stress and bring down inflammation – thus lowering your risk of cardiovascular problems. Needless to say, lifestyle modifications (such as weight loss, eating right, maintaining blood sugar levels and refraining from smoking) also improve overall quality of life.
Our next article in this series focuses on how to naturally reduce your cholesterol levels – with the help of right supplements and introducing relevant lifestyle changes. Stay tuned.
TV’s most likable dad died from sudden cardiac death last week. Alan Thicke’s massive heart attack was quick and unexpected and many of his closest friends have already come out saying there were no signs whatsoever. As tragic as this is, it’s equally upsetting to know the signs were likely present but no one was looking for them. Of course, this is not something that happens to just TV stars. Everyday people are taken from us in an instant. It’s an all too common scenario.
But it doesn’t have to be. Most specialists agree that heart disease is quite preventable and we now can put ourselves in a position of minimal risk. The key is to know what to look for.
Heart disease continues to be the leading cause of the death in this country and continues to be a threat in taking away those we love when we least expect it. It’s hard to overstate the impact that heart disease has in the US. Consider the following:
- Heart Disease affects 65 million Americans.
- 1 million Americans have heart attacks every year
- 1 person dies every 39 seconds from heart related problems
- 33-50% of deaths are due to heart related problems
- 33% of Americans have metabolic syndrome (a cluster of heart risk factors) associated with heart disease, diabetes, obesity and cancer)
- Total cost of heart disease in the US is estimated at $320 Billion.
To put that last statistic in perspective, the World Health Organization has estimated that ending world hunger would cost approximately $195 billion. It could be argued that the $320 billion spent on treating heart disease in a necessary expenditure, but studies suggest that 80-90% of heart disease is caused by modifiable diet and lifestyle factors; especially when you know the risks!
Herein lies the problem. Heart disease is one of the most misdiagnosed and mistreated conditions in medicine. Even though medical scientists have discovered so much about what causes heart disease over the past 20 years, the medical establishment is still operating on outdated science from 40-50 years ago. This includes testing and diagnostics. Hint: Normal cholesterol does not mean you don’t have heart disease.
We could spend hours covering all the research that’s been conducted by major universities. I’ll save you the time and provide a synopsis of facts published in some of the best peer reviewed journals in the world:
- High Cholesterol is not the primary cause of heart disease.
- Diets high in saturated fat and cholesterol don’t cause heart disease.
- Consumption of “heart healthy” vegetable oils is linked to heart disease, cancer and many other conditions
- Statin drugs don’t reduce the risk of death for most people, and have dangerous side effects and complications.
- Statin drugs can save lives for a very small percentage of the population, but they still cause complications.
Thicke, who was 69, was playing ice hockey with his son when he suffered his heart attack on Dec. 13. It happened with no symptoms.
For a heart attack to quickly lead to death, the damage to the heart needs to be great enough to cause the heart to beat irregularly and eventually stop entirely. The irregular heartbeats that result from the lack of oxygen start from the bottom of the heart, and aren't strong enough to generate blood flow,
When this occurs, the heart becomes very agitated and eventually the heartbeat becomes more of a quiver than strong pump. This is called fibrillation, and is what leads to less blood flowing which then causes the heart to stop.
Heart Attacks Don't Always Kill Instantly
It’s possible for the heart muscle to lead to irregular heartbeats long after the heart attack happens. A person can also die from a heart attack that causes no irregular heartbeat at all — the heart muscle can be so damaged from the lack of oxygen that the heart can no longer pump enough blood, which can lead to death. For death to occur immediately or shortly after a heart attack, there had to be a very large blockage that damaged a lot of the heart muscle.
Risk factors and symptoms
It's unclear what symptoms and risk factors Thicke had, if any, before his heart attack. My point is that we shouldn’t wait for symptoms to address them. Especially when it’s something as deadly as heart disease.
It’s widely accepted and most people know that risk factors of heart disease include high blood pressure, high cholesterol and a sedentary lifestyle. But this is literally the tip of the iceberg.
Things you should consider to Get a Grip On Predicting Heart Attacks & Sudden Death:
- Family History
- Genetic Testing
- Biometrics (waist to hip ratio, BMI, visceral fat, muscle mass)
- Vitals (blood pressure
- Blood chemistry (Cholesterol, Total, HDL, LDL, HDL-P, LDL-P, HS-CRP, ApoA, ApoB)
- EKG and Stress Testing
- CIMT ultrasound testing
Functional Medicine Assessments & Consultations
Beginning January 1st, I will start offering structured Functional Medicine consults. This will be separate from Family Health Chiropractic and something that is being launched nationally. That said, because you are patients of mine already, I’d like to give you priority over anyone else. I’d also like to offer you a deep discount on this service.
My Functional Medicine consults will be virtual, meaning we can meet via skype or phone and everything including paperwork, testing and diagnostics, treatment and protocols will be handled via my private patient portal. Because my time is already limited, I have to limit the number of patients I accept. NOTE: I promise, these spots will fill up fast so don’t delay. For those of you who don’t get registered, you’ll be put on a waiting list.
To learn more visit here: www.drdaniel.com/consult (Use coupon code: FAMILY for $200 off until spots are filled)
How will this work?
Step 1: Purchase your High Performance Health Assessment & Consultation
As a patient of mine you will be getting a deep discount compared to what everyone else will pay. Merry Christmas 🙂
Step 2: After purchasing your consult you can register at my private portal where you will create a personal account so I can communicate with you securely.
The initial forms you will fill out are likely more comprehensive than anything you’ve experienced before. This should give you an idea of how serious I am about your health. You probably have never had a consult with a doctor like this before!
After you finish filling out the forms, we will schedule a virtual consult and I will begin working on your case. I will identify 3 problem areas that we can begin addressing to improve health and work towards High Performance.
Step 3: You will get a follow up consultation containing specific functional medicine labs from the best laboratories in America along with specific action steps to take.
Everyone is going to be interested in improving their health at the beginning of the year. I don’t want to just improve health, I want to focus on High Performance health and this requires something different.
To learn more visit here: www.drdaniel.com/consult (Use coupon code: FAMILY for $200 off until spots are filled)
What is Functional Medicine?
In conventional medicine, there’s a doctor for every part of your body: cardiologists for the heart, gastroenterologists for the digestive system, neurologists for the brain and nervous system, podiatrists for your feet, ophthalmologists for your eyes and yes, chiropractic for your spine and acupuncturists for your points J
Due to this siloing, conventional medicine practitioners focus on individual body systems, rather than the whole person to understand the interrelated causes underlying disease and chronic illness. It looks at symptoms in order to name a disease and find a corresponding drug.
Alternative medicine is certainly more holistic and focused on whole body systems, but it usually falls short in diagnostics and objective testing. Alternative medicine practitioners (massage therapy, nutritionists, chiropractors, acupuncturists, homeopaths, naturopaths, etc) offer treatments that are much safer but not specific.
In functional medicine, we see the body as an interconnected whole, within a larger environment. We recognize that in order to treat one part of the body, all other parts must also be considered. This breaks apart artificial divisions of the body. Functional medicine looks at underlying phenomena that occur across specialties (inflammation, oxidative stress, toxicities, cellular energy problems, etc.) in order to understand the root cause of disease and find the right tools, at the right time, individualized for each person.
The table below compares eight key distinctions between functional and conventional medicine.
If you’re interested in High Performance Health or want to address any risk factors you may have regarding Heart Disease, Diabetes, Immunity, Gut Disturbance, Hormone Imbalances and/or Nutritional Issues, then please consider my functional medicine consults. Spots are limited, so act fast.
To learn more visit here: www.drdaniel.com/consult (Use coupon code: FAMILY for $200 off until spots are filled)